SLC25A12 inhibits Japanese encephalitis virus replication by interacting with the NS1 and enhancing the type I interferon response-Reference-Cited by-同舟云学术

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SLC25A12 inhibits Japanese encephalitis virus replication by interacting with the NS1 and enhancing the type I interferon response

Published:2024-10 Issue: Volume:297 Page:110199
ISSN:0378-1135
Container-title:Veterinary Microbiology
language:en
Short-container-title:Veterinary Microbiology

Author:

Yin You-qin,Liu Le-le,Jiang Yu-ting,Xing Jin-chao,Qi Wen-bao,Huang Li-hong

Funder

Basic and Applied Basic Research Foundation of Guangdong Province

Ministry of Science and Technology of the People's Republic of China

National Natural Science Foundation of China

National Key Research and Development Program of China

Publisher

Elsevier BV

Reference41 articles.

1. Flavivirus NS1 structures reveal surfaces for associations with membranes and the immune system;Akey;Science,2014

2. AGC1/2, the mitochondrial aspartate-glutamate carriers;Amoedo;Biochim Biophys. Acta,2016

3. Overexpression of the malate-aspartate NADH shuttle member Aralar1 in the clonal beta-cell line BRIN-BD11 enhances amino-acid-stimulated insulin secretion and cell metabolism;Bender;Clin. Sci.,2009

4. The Dengue Virus Nonstructural Protein 1 (NS1) Interacts with the Putative Epigenetic Regulator DIDO1 to Promote Flavivirus Replication in Mosquito Cells;Caraballo;J. Virol.,2022

5. Flavivirus genome organization, expression, and replication;Chambers;Annu. Rev. Microbiol.,1990

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