Cell entry of Bovine herpesvirus-1 through clathrin- and caveolin-mediated endocytosis requires activation of PI3K-Akt-NF-κB and Ras-p38 MAPK pathways as well as the interaction of BoHV-1 gD with cellular receptor nectin-1-Reference-Cited by-同舟云学术

Cell entry of Bovine herpesvirus-1 through clathrin- and caveolin-mediated endocytosis requires activation of PI3K-Akt-NF-κB and Ras-p38 MAPK pathways as well as the interaction of BoHV-1 gD with cellular receptor nectin-1

Published:2023-04 Issue: Volume:279 Page:109672
ISSN:0378-1135
Container-title:Veterinary Microbiology
language:en
Short-container-title:Veterinary Microbiology

Author:

Liu Yang,Zhang Qi,Zou Min,Cui Jin,Shi Xiaojie,Li Linjie,Wu Faxing,Xu Xingang

Funder

Key Research and Development Projects of Shaanxi Province

Publisher

Elsevier BV

Subject

General Veterinary,General Medicine,Microbiology

Reference39 articles.

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3. HSV activates Akt to trigger calcium release and promote viral entry: novel candidate target for treatment and suppression;Cheshenko;FASEB J.,2013

4. Entry of the Varicellovirus Canid herpesvirus 1 into Madin-Darby canine kidney epithelial cells is pH-independent and occurs via a macropinocytosis-like mechanism but without increase in fluid uptake;Eisa;Cell. Microbiol.,2021

5. Lipid-mediated endocytosis;Ewers;Cold Spring Harb. Perspect. Biol.,2011

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