Endogenous components of the striatum confer dopamine-sensitivity upon adenylate cyclase activity: The role of endogenous guanyl nucleotides

Author:

Chen Tai C.,Cote Thomas E.,Kebabian John W.

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference16 articles.

1. Saturation assay for cyclic AMP using endogenous binding protein;Brown,1972

2. Mechanism of adenylate cyclase activation through the β-adrenergic receptor: catecholamine-induced displacement of bound GDP by GTP;Cassel,1978

3. Endogenous GTP and the regulation of epinephrine stimulation of adenylate cyclase;Clark;J. cyclic Nucleotide Res.,1978

4. Characterization of a dopamine-sensitive adenylate cyclase in the rat caudate nucleus;Clement-Cormier;J. Neurochem.,1975

5. Dopamine-sensitive adenylate cyclase from the rat caudate nucleus: Regulation by guanyl nucleotides and the interaction of magnesium and magnesium ATP;Clement-Cormier;J. Neurochem.,1978

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1. Site of Pertussis Toxin-Induced ADP-Ribosylation on Gi Is Critical for Receptor Modulation of GDP Interaction with Gi;Molecular Endocrinology;1989-02

2. Dopaminergic Neurons: Basic Aspects;Catecholamines II;1989

3. The D-1 Dopamine Receptor;Advances in Experimental Medicine and Biology;1988

4. Dopamine Receptors And Signal Transduction;Progress in Parkinson Research;1988

5. Dopamine stimulated increase of GTP levels in the rat retina.;Journal of Biological Chemistry;1985-04

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