Pertussis toxin lesioning of the nucleus caudate-putamen attenuates adenylate cyclase inhibition and alters neuronal electrophysiological activity

Author:

Boyajian Christine L.,Bickford-Wimer Paula,Kim Martha B.,Freedman Robert,Cooper Dermot M.F.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference35 articles.

1. Quantitative autoradiographic localization of the D1 and D2 subtypes of dopamine receptors in rat brain;Boyson;J. Neuroscience,1986

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3. Guanine nucleotide regulatory proteins and signal transduction in the CNS—Multiple mechanisms;Cooper;Clin. Neuropharmacol.,1986

4. Dopamine, acting through D2 receptors, inhibits rat striatal adenylate cyclase by a GTP-dependent process;Cooper;Mol. Pharmacol.,1985

5. Adenosine A1 receptors inhibit both adenylate cyclase activity and TRH-activated Ca2+ channels by a pertussis toxin-sensitive mechanism in GH3 cells;Cooper;Cell. Signalling,1989

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