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Body region shocked need not critically define the neurochemical basis of stress analgesia

  • Published:1984-12 Issue:2 Volume:323 Page:316-319
  • ISSN:0006-8993
  • Container-title:Brain Research
  • language:en
  • Short-container-title:Brain Research

Author:

Cannon J. Timothy,Terman Gregory W.,Lewis James W.,Liebeskind John C.

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference22 articles.

1. Stress-induced increase in endogenous opiate peptides; concurrent analgesia and its partial reversal by naloxone;Akil,1976

2. Dose-dependent reductions by naloxone of analgesia induced by cold-water stress;Bodnar;Pharmacol. Biochem. Behav.,1978

3. Differential effects of naloxone on analgesia and vocalizations induced by brief durations of pulsed footshock;Cannon;Soc. Neurosci. Abstr.,1983

4. Autoanalgesia: opiate and non-opiate mechanisms;Chance;Neurosci. Biobehav. Rev.,1980

5. A method for determinating loss of pain sensation;D'Amour;J. Pharmacol. exp. Ther.,1941

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1. Pain control in tonic immobility (TI) and other immobility models;Progress in Brain Research;2022

2. Stress and Opioid Systems;Hormones, Brain and Behavior;2017

3. Stress, Opioid Peptides, and Their Receptors;Hormones, Brain and Behavior;2009

4. High intensity magnetic stimulation over the lumbosacral spine evokes antinociception in rats;Clinical Neurophysiology;2002-07

5. Stress, Opioid Peptides, and Their Receptors;Hormones, Brain and Behavior;2002
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