Kainic acid neurotoxicity in the goldfish optic tectum No protection by the NMDA receptor antagonist MK801 and partial protection by abolition of a tectal excitatory input
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience
Reference33 articles.
1. Anticonvulsant activity of excitatory amino acids antagonists;Chapman,1988
2. Pharmacology of glutamate neurotoxicity in cortical cell culture: attenuation by NMDA antagonists;Choi;J. Neurosci.,1988
3. Ketamine, phencyclidine and MK801 protect against kainic acid-induced, seizure-related brain damage;Clifford;Epilepsia,1990
4. Patterns of neurotransmitter function in the optic tectum of teleosts;Contestabile;Adv. Physiol. Sci.,1981
5. Pharmacological manipulation of GABA system does not protect the goldfish optic tectum from the neuroexcitatory and neurotoxic action of kainic acid;Contestabile;Brain Res.,1983
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Nmda antagonists: Antiepileptic-neuroprotective drugs with diversified neuropharmacological profiles;Pharmacological Research;1995-07
2. 6,7-Dinitroquinoxaline-2,3-dione but not MK-801 exerts a protective effect against kainic acid neurotoxicity in the goldfish retina;Neuroscience Letters;1995-06
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