Long-term caffeine treatment leads to a decreased susceptibility to NMDA-induced clonic seizures in mice without changes in adenosine A1 receptor number-Reference-Cited by-同舟云学术

Long-term caffeine treatment leads to a decreased susceptibility to NMDA-induced clonic seizures in mice without changes in adenosine A1 receptor number

Published:1993-05 Issue:1-2 Volume:612 Page:271-277
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Georgiev Vasil,Johansson Bjo¨rn,Fredholm Bertil B.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference35 articles.

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2. Changes in seizure latency correlate with alterations in A1 adenosine receptor binding during daily repeated pentylenetetrazol-induced convulsions in different mouse brain areas;Angelatou;Neurosi. Lett.,1991

3. Adenosine inhibits epileptiform activity arising in hippocampal area CA3;Ault;Br. J. Pharmacol.,1986

4. Anticonvulsant effects of adenosine analogs on amygdaloid-kindled seizures in rats;Barraco;Neurosci. Lett.,1984

5. Chronic caffeine consumption increases the number of brain adenosine receptors;Boulenger;Life Sci.,1983

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