Modulation of adenylate cyclase activity of mouse spinal cord-ganglion explants by opioids, serotonin and pertussis toxin

Author:

Makman Maynard H.,Dvorkin B.,Crain Stanley M.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference57 articles.

1. Attali, B. and Vogel, Z., Inhibition of adenylate cyclase and induction of heterologous desentization by kappa agonists in rat spinal cord. In J.W. Holaday A. Herz and P.-Y. Law (Eds.), Progress in Opioid Research, Proc. Int. Narcotics Res. Conf., National Institute on Drug Abuse Research Monograph no. 75, U.S. Gov. Printing Office, Washington, DC, pp. 137–140.

2. Coupling of opiate receptors to adenylate cyclase: requirement for Na+ and GTP;Blume,1979

3. Purification and properties of the inhibitory guanine nucleotide-binding regulatory component of adenylate cyclase;Bogoch;J. Biol. Chem.,1984

4. Maturation of opioid sensitivity of fetal mouse dorsal-root ganglion neuron perikarya in organotypic cultures: regulation by spinal cord;Chalazonitis;Neuroscience,1986

5. Inhibition of cyclic AMP-dependent protein kinase blocks opioid prolongation of the action potential of dorsal root ganglia (DRG) neurons;Chen;Soc. Neurosci. Abstr.,1987

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