AV3V lesion suppresses the pressor, dipsogenic and natriuretic responses to cholinergic activation of the septal area in rats

Author:

Colombari Eduardo,Abra˜o Saad Wilson,de Arruda Camargo Luiz Antonio,Renzi Antonio,De Luca Laurival Antonio,Vanderlei Menani José

Publisher

Elsevier BV

Subject

Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience

Reference21 articles.

1. Role of the hypothalamus in the control of atrial natriuretic peptide release;Baldissera,1989

2. Sodium excretion following lesions of preoptic recess periventricular tissue in the rats;Bealer;Am. J. Physiol.,1983

3. Preoptic hypothalamic periventricular lesions reduce natriuresis to volume expansion;Bealer;Am. J. Physiol.,1983

4. Mechanisms of altered sodium excretion after preoptic hypothalamic lesions;Bealer;Am. J. Physiol.,1983

5. The role of the anteroventral third ventricle (AV3V) region in experimental hypertension;Brody;Circ. Res.,1978

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