Decreased norepinephrine in the ventral lamina terminalis region is associated with angiotensin II drinking response deficits following local 6-hydroxydopamine injections-Reference-Cited by-同舟云学术

Decreased norepinephrine in the ventral lamina terminalis region is associated with angiotensin II drinking response deficits following local 6-hydroxydopamine injections

Published:1989-02 Issue:1-2 Volume:480 Page:65-71
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Thomas Cunningham J.,Johnson Alan Kim

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference34 articles.

1. Periventricular noradrenergic systems are critical for angiotensin-induced drinking and blood pressure responses;Bellin;Brain Research,1987

2. Localized injections of 6-hydroxydopamine into lamina terminalis-associated structures: effects on experimentally induced drinking and pressor responses;Bellin;Brain Research,1987

3. Selective catecholamine depletion of structures along ventral lamina terminalis: effects on experimentally-induced drinking and pressor responses;Bellin;Brain Research,1988

4. Inhibiting the rabbit caudal ventrolateral medulla prevents baroreceptor-initiated secretion of vasopressin;Blessing;J. Physiol. (Lond.),1985

5. Excitation of neuronal function in rabbit caudal ventrolateral medulla elevates plasma vasopressin;Blessing;Neurosci. Lett.,1985

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