Limited responsiveness of LHRH neurons norepinephrine may account for failure of locus coeruleus or medullary A1 electrical stimulation to increase plasma LH in estrogen-treated ovariectomized rats-Reference-Cited by-同舟云学术

Limited responsiveness of LHRH neurons norepinephrine may account for failure of locus coeruleus or medullary A1 electrical stimulation to increase plasma LH in estrogen-treated ovariectomized rats

Published:1989-01 Issue:1 Volume:476 Page:35-44
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Hartman Richard D.,Petersen Sandra,Barraclough Charles A.

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference32 articles.

1. Evidence for γ-aminobutyric acid modulation of ovarian hormonal effects on luteinizing hormone secretion and hypothalamic catecholamine activity in the female rat;Adler;Endocrinology,1986

2. A priming effect of luteinizing hormone releasing factor on the anterior pituitary gland in the female rat;Aiyer;J. Endocrinol.,1979

3. The role of catecholamines in the regulation of pituitary LH and FSH secretion;Barraclough;Endocr. Rev.,1982

4. Preoptic catecholamine, GABA, and glutamate release in ovariectomized and ovariectomized estrogen-treated rats utilizing a push-pull cannula technique;Demling;Neuroendocrinology,1985

5. Differential regulation of pulsatile luteinizing hormone (LH) and follicle stimulating hormone secretion in ovariectomized rats disclosed by treatment with a LH-releasing hormone antagonist and phenobarbital;DePaolo;Endocrinology,1985

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