Involvement of complement in degeneration of sympathetic nerves after administration of antiserum to dopamine β-hydroxylase
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Clinical Neurology,Molecular Biology,General Neuroscience
Reference20 articles.
1. Histochemical evidence for the degeneration of peripheral noradrenergic axons following intravenous injection of antibodies to dopamine β-hydroxylase;Costa;Neurosci. Lett.,1976
2. Selective uptake and retrograde axonal transport of dopamine β-hydroxylase antibodies in peripheral adrenergic neurons;Fillenz;Brain Research,1976
3. The use of glyoxylic acid for the fluorescence histochemical demonstration of peripheral stores of noradrenaline and 5-hydroxytryptamine in whole mounts;Furness;Histochemistry,1975
4. Synaptic vesicles in sympathetic neurons;Geffen;Physiol. Rev.,1971
5. Fine structure and fluorescence morphology of adrenergic nerves after 6-hydroxydopamine in vivo and in vitro;Ho¨kfelt;Z. Zellforsch.,1972
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1. Complement-mediated neurotoxicity is regulated by homologous restriction;Brain Research;1995-02
2. Lesions in the hypothalamus after active immunisation against GnRH in the pig;Journal of Neuroimmunology;1993-10
3. Death of intermediolateral spinal cord neurons follows selective, complement-mediated destruction of peripheral preganglionic sympathetic terminals by acetylcholinesterase antibodies;Neuroscience;1993-05
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5. Antibodies to Neuropeptides: Biological Effects and Mechanisms of Action;Advances in Experimental Medicine and Biology;1990
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