Is substance P a primary afferent neurotransmitter for nociceptive input? III. Valproic acid and chlordiazepoxide decrease behaviors elicited by intrathecal injection of substance P and excitatory compounds
Author:
Publisher
Elsevier BV
Subject
Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience
Reference49 articles.
1. Intrathecal (d-Pro2,d-Trp7.9)-SP elicits hypoalgesia and motor blockade in the rat and antagonizes noxious responses induced by substance P;Akerman;Acta Physiol. Scand.,1982
2. Inhibition of intrathecally administered picrotoxin and bicuculline-induced convulsions in mice by pipecolic acid or GABA;Beitz;Eur. J. Pharmacol.,1985
3. Hyperalgesia induced by altered glycinergic activity at the spinal cord;Beyer;Life Sci.,1985
4. Is substance P a primary afferent neurotransmitter for nociceptive input? II. Spinalization does not reduce and intrathecal morphine potentiates behavioral responses to substance P;Bossut;Brain Research,1988
5. Ultrastructure of chemically defined neuron systems in the dorsal horn of the monkey. I. Substance P immunoreactivity;De Lanerolle;Brain Research,1983
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2. The promise of substance P inhibitors in fibromyalgia;Rheumatic Disease Clinics of North America;2002-05
3. Spinal cord substance P receptor immunoreactivity increases in both inflammatory and nerve injury models of persistent pain;Neuroscience;1996-01
4. Infusion of substance P or neurokinin A by microdialysis alters responses of primate spinothalamic tract neurons to cutaneous stimuli and to iontophoretically released excitatory amino acids;Pain;1995-06
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