Depolarization-evoked release of glutamate, aspartate and γ-aminobutyric acid from rat dorsal spinal cord does not originate from capsaicin-sensitive neurons-Reference-Cited by-同舟云学术

Depolarization-evoked release of glutamate, aspartate and γ-aminobutyric acid from rat dorsal spinal cord does not originate from capsaicin-sensitive neurons

Published:1991-08 Issue:2 Volume:555 Page:332-334
ISSN:0006-8993
Container-title:Brain Research
language:en
Short-container-title:Brain Research

Author:

Donnerer Josef

Publisher

Elsevier BV

Subject

Developmental Biology,Neurology (clinical),Molecular Biology,General Neuroscience

Reference17 articles.

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3. Glutamate and substance P coexist in primary afferent terminals in the superficial laminae of spinal cord;De Biasi,1988

4. Capsaicin and nociception in the rat and mouse. Possible role of substance P;Gamse;Naunyn-Schmiedeberg's Arch. Pharmacol.,1982

5. Glutamate and aspartate immunoreactivity in corticospinal neurons of rats;Giuffrida;J. Comp. Neurol.,1989

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1. GABA transporter type 1 (GAT-1) uptake inhibition reduces stimulated aspartate and glutamate release in the dorsal spinal cord in vivo via different GABAergic mechanisms;Neuropharmacology;2007-12

2. Nociceptive transmitter release in the dorsal spinal cord by capsaicin-sensitive fibers after noxious gastric stimulation;Brain Research;2005-03

3. Neonatal capsaicin treatment abolishes formalin-induced spinal PGE2 release;NeuroReport;1997-07

4. GABAB Receptor Control of Neurotransmitter Release in Mammalian Brain: Modification During Chronic Inflammation;Neutrotransmitter Release and Uptake;1997

5. Evidence for release of glutamic acid, aspartic acid and substance P but not γ-aminobutyric acid from primary afferent fibres in rat spinal cord;European Journal of Pharmacology;1996-04