Molecular basis of the inner blood-retinal barrier and its breakdown in diabetic macular edema and other pathological conditions-Reference-Cited by-同舟云学术

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Molecular basis of the inner blood-retinal barrier and its breakdown in diabetic macular edema and other pathological conditions

Published:2013-05 Issue: Volume:34 Page:19-48
ISSN:1350-9462
Container-title:Progress in Retinal and Eye Research
language:en
Short-container-title:Progress in Retinal and Eye Research

Author:

Klaassen Ingeborg,Van Noorden Cornelis J.F.,Schlingemann Reinier O.

Funder

Diabetes Fonds Nederland

Landelijke Stichting voor Blinden en Slechtzienden

Stichting Blinden-Penning

Stichting Oogfonds Nederland

Vereniging Blindenbelangen Rotterdam

Stichting Blindenhulp

Prof. Hoppenbrouwers Fonds

Stichting Nederlands Oogheelkundig Onderzoek

Publisher

Elsevier BV

Subject

Sensory Systems,Ophthalmology

Reference434 articles.

1. Inflammatory mediators and modulation of blood–brain barrier permeability;Abbott;Cell. Mol. Neurobiol.,2000

2. Cytokines in the vitreous of patients with proliferative diabetic retinopathy;Abu el Asrar;Am. J. Ophthalmol.,1992

3. Immunological mechanisms in the pathogenesis of diabetic retinopathy;Adamis;Semin. Immunopathol.,2008

4. Suppression of retinal neovascularization in vivo by inhibition of vascular endothelial growth factor (VEGF) using soluble VEGF-receptor chimeric proteins;Aiello;Proc. Natl. Acad. Sci. U.S.A.,1995

5. Vascular endothelial growth factor-induced retinal permeability is mediated by protein kinase C in vivo and suppressed by an orally effective beta-isoform-selective inhibitor;Aiello;Diabetes,1997

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