Realistic concentrations of Bisphenol-A trigger a neurotoxic response in the brain of zebrafish: Oxidative stress, behavioral impairment, acetylcholinesterase inhibition, and gene expression disruption

Author:

Heredia-García Gerardo,Elizalde-Velázquez Gustavo Axel,Gómez-Oliván Leobardo ManuelORCID,Islas-Flores Hariz,García-Medina Sandra,Galar-Martínez Marcela,Dublán-García Octavio

Funder

Consejo Nacional de Ciencia y Tecnología

Publisher

Elsevier BV

Subject

General Medicine,General Chemistry,Environmental Chemistry,Environmental Engineering,Pollution,Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference47 articles.

1. Bisphenol A induces histopathological, hematobiochemical alterations, oxidative stress, and genotoxicity in common carp (Cyprinus carpio L.);Afzal,2022

2. ‘Glycogen Synthase Kinase 3 Is Part of the Molecular Machinery Regulating the Adaptive Response to LPS Stimulation in Microglial Cells’, Brain, Behavior, and Immunity;Ajmone-Cat,2016

3. Evaluation of oxidative stress, antioxidant enzymes and genotoxic potential of bisphenol A in fresh water bighead carp (Aristichthys nobils) fish at low concentrations;Akram;Environ. Pollut.,2021

4. Acetylcholinesterase is required for neuronal and muscular development in the zebrafish embryo;Behra;Nat. Neurosci.,2002

5. Adult neurogenesis in the female mouse hypothalamus: estradiol and high-fat diet alter the generation of newborn neurons expressing estrogen receptor α;Bless;eNeuro,2016

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