Dual loss of succinate dehydrogenase (SDH) and complex I activity is necessary to recapitulate the metabolic phenotype of SDH mutant tumors-Reference-Cited by-同舟云学术

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Dual loss of succinate dehydrogenase (SDH) and complex I activity is necessary to recapitulate the metabolic phenotype of SDH mutant tumors

Published:2017-09 Issue: Volume:43 Page:187-197
ISSN:1096-7176
Container-title:Metabolic Engineering
language:en
Short-container-title:Metabolic Engineering

Author:

Lorendeau Doriane,Rinaldi Gianmarco,Boon Ruben,Spincemaille Pieter,Metzger Kristine,Jäger Christian,Christen Stefan,Dong Xiangyi,Kuenen Sabine,Voordeckers Karin,Verstreken Patrik,Cassiman David,Vermeersch Pieter,Verfaillie Catherine,Hiller Karsten,Fendt Sarah-Maria

Funder

CIG, European Union

FWO

Concern Foundation

FWO – Research Grants, Belgium

KU-Leuven

Eugène Yourassowsky Schenking

Bayer Health Care

Publisher

Elsevier BV

Subject

Applied Microbiology and Biotechnology,Bioengineering,Biotechnology

Reference46 articles.

1. Depression of neuron responses to acetylcholine by combined application of norepinephrine and substrates of the tricarboxylic acid cycle;Andreev;Cell Mol. Neurobiol.,1986

2. Elementary metabolite units (EMU): a novel framework for modeling isotopic distributions;Antoniewicz;Metab. Eng.,2007

3. Late-onset optic atrophy, ataxia, and myopathy associated with a mutation of a complex II gene;Birch-Machin;Ann. Neurol.,2000

4. An essential role of the mitochondrial electron transport chain in cell proliferation is to enable aspartate synthesis;Birsoy;Cell,2015

5. Mutation of a nuclear succinate dehydrogenase gene results in mitochondrial respiratory chain deficiency;Bourgeron;Nat. Genet.,1995

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