An ‘anatomical cascade hypothesis’ for Alzheimer's disease-Reference-Cited by-同舟云学术

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An ‘anatomical cascade hypothesis’ for Alzheimer's disease

Published:1992-06 Issue:6 Volume:15 Page:200-201
ISSN:0166-2236
Container-title:Trends in Neurosciences
language:en
Short-container-title:Trends in Neurosciences

Author:

Hardy John

Publisher

Elsevier BV

Subject

General Neuroscience

Reference26 articles.

1. Amyloid deposition as the central event in the aetiology of Alzheimer's disease

2. Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease

3. Early-onset Alzheimer's disease caused by mutations at codon 717 of the β-amyloid precursor protein gene

4. A mutation in the Amyloid Precursor Protein Associated with Hereditary Alzheimer's Disease

5. The Neuroanatomy of Alzheimer's Disease

Cited by 75 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Chemical signatures delineate heterogeneous amyloid plaque populations across the Alzheimer’s disease spectrum;2024-06-03

2. The heritability of blood‐based biomarkers related to risk of Alzheimer's disease in a population‐based sample of early old‐age men;Alzheimer's & Dementia;2023-08-25

3. Tetramodal Chemical Imaging Delineates the Lipid–Amyloid Peptide Interplay at Single Plaques in Transgenic Alzheimer’s Disease Models;Analytical Chemistry;2023-03-01

4. Stable cerebrospinal fluid neurogranin and β-site amyloid precursor protein cleaving enzyme 1 levels differentiate predementia Alzheimer’s disease patients;Brain Communications;2022-09-01

5. The genetic and environmental etiology of blood-based biomarkers related to risk of Alzheimer’s Disease in a population-based sample of early old-age men;2022-08-11

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