Novel PGK1 determines SKP2-dependent AR stability and reprograms granular cell glucose metabolism facilitating ovulation dysfunction

Author:

Liu Xia,Sun Changfa,Zou Kexin,Li Cheng,Chen Xiaojun,Gu Hangchao,Zhou Zhiyang,Yang Zuwei,Tu Yaoyao,Qin Ningxin,Zhao Yiran,Wu Yimei,Meng Yicong,Ding Guolian,Liu Xinmei,Sheng Jianzhong,Yu Chuanjin,Huang HefengORCID

Funder

National Key Research and Development Program of China

International Cooperation Project of China and Canada NSFC

National Natural Science Foundation of China

Chinese Academy of Medical Sciences Research Unit

CAMS Inovation Fund for Medical Sciences

The Interdisciplinary Key Program of Shanghai Jiao Tong University

Shen Kang Three Year Action plan

postdoctoral

Shanghai Municipal Key Clinical Specialty, Shanghai, China

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference57 articles.

1. Polycystic ovary syndrome;Norman;Lancet,2007

2. Polycystic ovary syndrome: definition, aetiology, diagnosis and treatment;Escobar-Morreale;Nat Rev Endocrinol,2018

3. Phosphorylation of c-Fos by members of the p38 MAPK family. Role in the AP-1 response to UV light;Tanos;J. Biol. Chem.,2005

4. Regulation of AP1 (Jun/Fos) factor expression and activation in ovarian granulosa cells. Relation of JunD and Fra2 to terminal differentiation;Sharma;J. Biol. Chem.,2000

5. The role of Jun, Fos and the AP-1 complex in cell-proliferation and transformation;Angel;Biochim. Biophys. Acta,1991

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