DsbA-L interacts with VDAC1 in mitochondrion-mediated tubular cell apoptosis and contributes to the progression of acute kidney disease-Reference-Cited by-同舟云学术

DsbA-L interacts with VDAC1 in mitochondrion-mediated tubular cell apoptosis and contributes to the progression of acute kidney disease

Published:2022-02 Issue: Volume:76 Page:103859
ISSN:2352-3964
Container-title:eBioMedicine
language:en
Short-container-title:eBioMedicine

Author:

Li Xiaozhou,Pan Jian,Li Huiling,Li Guangdi^ORCID,Liu Bohao,Tang Xianming,Liu Xiangfeng,He Zhibiao,Peng Zhenyu,Zhang Hongliang,Wang Luxiang,Li Yijian,Xiang Xudong,Chai Xiangping,Yuan Yunchang,Zheng Peilin,Zhang Dongshan^ORCID

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference48 articles.

1. Acute kidney injury;Zuk;Annu Rev Med,2016

2. Activation of hepatocyte growth factor receptor, c-met, in renal tubules is required for renoprotection after acute kidney injury;Zhou;Kidney Int,2013

3. Acute kidney injury following cardiac surgery: current understanding and future directions;O'Neal;Crit Care,2016

4. Mitochondria: a therapeutic target in acute kidney injury;Ishimoto;Nephrol Dial Transplant,2016

5. Regulated cell death in AKI;Linkermann;J Am Soc Nephrol,2014

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