Lyn kinase represses mucus hypersecretion by regulating IL-13-induced endoplasmic reticulum stress in asthma-Reference-Cited by-同舟云学术

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Lyn kinase represses mucus hypersecretion by regulating IL-13-induced endoplasmic reticulum stress in asthma

Published:2017-02 Issue: Volume:15 Page:137-149
ISSN:2352-3964
Container-title:EBioMedicine
language:en
Short-container-title:EBioMedicine

Author:

Wang Xing,Yang Xiaoqiong,Li Yin,Wang Xiaoyun,Zhang Yun,Dai Xi,Niu Bin,Wu Juan,Yuan Xiefang,Xiong Anjie,Liu Zhigang,Zhong Nanshan,Wu Min,Li Guoping

Funder

Chinese National Science Foundation

National Institute of Health

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Reference53 articles.

1. IL-13-induced airway mucus production is attenuated by MAPK13 inhibition;Alevy;J. Clin. Invest.,2012

2. Endoplasmic reticulum stress impairs IL-4/IL-13 signaling through C/EBPbeta-mediated transcriptional suppression;Arensdorf;J. Cell Sci.,2013

3. A dual PI3K/mTOR inhibitor, PI-103, cooperates with stem cell-delivered TRAIL in experimental glioma models;Bagci-Onder;Cancer Res.,2011

4. A fungal protease allergen provokes airway hyper-responsiveness in asthma;Balenga;Nat. Commun.,2015

5. IL13 receptor alpha2 signaling requires a scaffold protein, FAM120A, to activate the FAK and PI3K pathways in colon cancer metastasis;Bartolome;Cancer Res.,2015

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4. Fine particulate matter promotes airway inflammation and mucin production by activating endoplasmic reticulum stress and the IRE1α/NOD1/NF‑κB pathway;International Journal of Molecular Medicine;2023-08-28

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