Dynamic viral integration patterns actively participate in the progression of BK polyomavirus-associated diseases after renal transplantation

Author:

Wang YuchenORCID,Yan SushaORCID,Liu YannaORCID,Yan ZiyanORCID,Deng WenfengORCID,Geng JianORCID,Li ZhuolinORCID,Xia RenfeiORCID,Zeng WenliORCID,Zhao TingORCID,Fang YilingORCID,Liu NaORCID,Yang LinglingORCID,Cheng ZhongyiORCID,Xu JianORCID,Wu Chin-LeeORCID,Miao YunORCID

Funder

National Natural Science Foundation of China

National College Students Innovation and Entrepreneurship Training Program

Publisher

Elsevier BV

Subject

Pharmacology (medical),Transplantation,Immunology and Allergy

Reference53 articles.

1. BK polyomavirus: clinical aspects, immune regulation, and emerging therapies;Ambalathingal;Clin Microbiol Rev,2017

2. BK polyomavirus virus glomerular tropism: implications for virus reactivation from latency and amplification during immunosuppression;Alcendor;J Clin Med,2019

3. Quantification of plasma BK polyomavirus loads is affected by sequence variability, amplicon length, and non-encapsidated viral DNA genome fragments;Leuzinger;J Clin Virol,2019

4. BK virus infection in allogeneic hematopoietic cell transplantation: an update on pathogenesis, immune responses, diagnosis and treatments;Saade;J Infect,2020

5. The SV40 large T antigen-p53 complexes bind and activate the insulin-like growth factor-I promoter stimulating cell growth;Bocchetta;Cancer Res,2008

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