Impaired neurogenesis by methylazoxymethanol in newborn rats results in transient reduction of ornithine decarboxylase and polyamines in the cerebellum, but not in the olfactory bulbs
Author:
Affiliation:
1. Department of Biology; University of Bologna; 40126 Bologna Italy
Publisher
Wiley
Subject
Developmental Biology,Developmental Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/0736-5748(94)90091-4/fullpdf
Reference32 articles.
1. Autoradiographic and histological studies of postnatal neurogenesis. III. Dating the time of production and onset of differentiation of cerebellar microneurons in rats;Altman;J. comp. Neurol.,1969
2. Effects of early postnatal guanethidine administration on adrenal medulla and brain of developing rats;Bartolome;Biochem. Pharmacol.,1976
3. Impaired development of cerebellar cortex in rats treated postnatally with α-difluoromethylornithine;Bartolome;Neuroscience,1985
4. Induction of ornithine decarboxylase as a possible mediator of seizure-induced changes in genomic expression in rat hippocampus;Baudry;J. Neurosci.,1986
5. 3H-thymidine-radiographic studies of neurogenesis in the rat olfactory bulb;Bayer;Exp. Brain Res.,1983
Cited by 1 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Chronic neonatal blockade of NMDA receptor does not affect developmental polyamine metabolism but results in altered response to the excitotoxic induction of ornithine decarboxylase;Neurochemistry International;1994-06
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