OX62+OX6+OX35+ rat dendritic cells are unable to prime CD4+ T cells for an effective immune response following acute burn injury
Author:
Publisher
Elsevier BV
Subject
Immunology
Reference28 articles.
1. Thermal injury-plus-sepsis contributes to a substantial deletion of intestinal mesenteric lymph node CD4 T cell via apoptosis;Fazal;International Journal of Biological Sciences,2007
2. Antigen presenting cells (APCs) from thermally injured and/or septic rats modulate CD4 +T cell responses of naïve rat;Fazal;Biochimica et Biophysica Acta,2005
3. Burn injury triggered dysfunction in dendritic cell response to TLR9 activation and resulted in skewed T cell functions;Shen;PLoS One,2012
4. Dendritic cell depletion in burn patients is regulated by MafB expression;Williams;Journal of Burn Care and Research,2012
5. Selective effect of burn injury on splenic CD11c (+) dendritic cells and CD8alpha (+) CD4 (−) CD11c (+) dendritic cell subsets;Patenaude;Cellular and Molecular Life Sciences,2010
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3. Effects of IL-10 on OX62, MHC-II and CD86 in bone marrow DCs in rats with organophosphate poisoning;Experimental and Therapeutic Medicine;2017-12-14
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