Amyloid‐beta oligomerization is associated with the generation of a typical peptide fragment fingerprint-Reference-Cited by-同舟云学术

Amyloid‐beta oligomerization is associated with the generation of a typical peptide fragment fingerprint

Published:2016-04-26 Issue:9 Volume:12 Page:996-1013
ISSN:1552-5260
Container-title:Alzheimer's & Dementia
language:en
Short-container-title:Alzheimer's & Dementia

Author:

Rudinskiy Nikita¹,Fuerer Christophe²,Demurtas Davide³,Zamorano Sebastian²,De Piano Cyntia²,Herrmann Abigail G.⁴⁵⁶,Spires-Jones Tara L.⁴⁵⁶,Oeckl Patrick⁷,Otto Markus⁷,Frosch Matthew P.⁸,Moniatte Marc²,Hyman Bradley T.¹,Schmid Adrien W.²

Affiliation:

1. Department of Neurology, Alzheimer's Disease Research Laboratory, MassGeneral Institute for Neurodegenerative Disease, Massachusetts General Hospital Harvard Medical School Charlestown MA USA

2. School of Life Sciences, Proteomics Core Facility Ecole Polytechnique Fédérale de Lausanne (EPFL) Lausanne Switzerland

3. School of Basic Sciences, Interdisciplinary Centre for Electron Microscopy (CIME) Ecole Polytechnique Fédérale de Lausanne (EPFL) Lausanne Switzerland

4. Center for Cognitive and Neural Systems The University of Edinburgh Edinburgh Scotland

5. Centre for Dementia Prevention The University of Edinburgh Edinburgh Scotland

6. Euan MacDonald Centre for Motorneurone Disease The University of Edinburgh Edinburgh Scotland

7. Department of Neurology Ulm University Hospital Ulm Germany

8. Massachusetts General Hospital and Harvard Medical School Massachusetts General Institute for Neurodegenerative Disease Charlestown MA USA

Funder

École Polytechnique Fédérale de Lausanne

Alzheimer’s Research UK

Alzheimer's Society

Massachusetts Alzheimer Disease Research Center

Alzheimer's Research UK (ARUK-SPG2013-1)

Wellcome Trust Institutional Strategic Support Fund

Publisher

Wiley

Subject

Psychiatry and Mental health,Cellular and Molecular Neuroscience,Geriatrics and Gerontology,Neurology (clinical),Developmental Neuroscience,Health Policy,Epidemiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1016/j.jalz.2016.03.011

Reference49 articles.

1. Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivo

2. Effects of secreted oligomers of amyloid β-protein on hippocampal synaptic plasticity: a potent role for trimers

3. Certain Inhibitors of Synthetic Amyloid -Peptide (A ) Fibrillogenesis Block Oligomerization of Natural A and Thereby Rescue Long-Term Potentiation

4. Cognitive effects of cell-derived and synthetically derived Aβ oligomers

5. A specific amyloid-β protein assembly in the brain impairs memory

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