The uremic toxin methylguanidine increases the oxidative metabolism and accelerates the apoptosis of canine neutrophils-Reference-Cited by-同舟云学术

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The uremic toxin methylguanidine increases the oxidative metabolism and accelerates the apoptosis of canine neutrophils

Published:2017-03 Issue: Volume:185 Page:14-19
ISSN:0165-2427
Container-title:Veterinary Immunology and Immunopathology
language:en
Short-container-title:Veterinary Immunology and Immunopathology

Author:

Bosco A.M.,Almeida B.F.M.,Pereira P.P.,dos Santos D.B.,Neto Á.J.S.,Ferreira W.L.,Ciarlini P.C.

Funder

São Paulo Research Foundation (FAPESP)

Publisher

Elsevier BV

Subject

General Veterinary,Immunology

Reference32 articles.

1. Neutrophil dysfunction varies with the stage of canine visceral leishmaniosis;Almeida;Vet. Parasitol.,2013

2. Leishmaniasis causes oxidative stress and alteration of oxidative metabolism and viability of neutrophils in dogs;Almeida;Vet. J.,2013

3. Significance of reactive oxygen in kidney disease elucidated by uremic toxins;Aoyagi;J. Artif. Organs,2001

4. Free p-cresol alters neutrophil function in dogs;Bosco;Artif. Organs,2016

5. Production of methylguanidine in dogs with acute and chronic renal failure;Brooks;Clin. Sci. (Lond.),1989

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