Acetylcholine contractures of skeletal muscles: Inhibition by chlorpromazine and diltiazem
Author:
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Pharmacology
Reference21 articles.
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2. Voltage dependencies of the effects of chlorpromazine on the nicotinic receptor channel from mouse muscle cell line Sol8;Benoit;Neurosci. Lett.,1993
3. The effect of the benzothiazepine diltiazem on force and Ca2+ current in isolated frog skeletal muscle fibres;Bohle;J. Physiol, Lond.,1992
4. The voltage-activation of contraction in skeletal muscle;Dulhunty;Prog. Biophys. Molec. Biol.,1992
5. Acetylcholine regulation of nicotinic receptor channels through a putative G protein in chick myotubes;Eusebi;J. Physiol, Lond.,1987
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Inhibition of human α1E subunit-mediated Ca2+ channels by the antipsychotic agent chlorpromazine;Acta Physiologica Scandinavica;2001-12
2. Sympathoadrenal Hyperactivity and the Etiology of Neuroleptic Malignant Syndrome;American Journal of Psychiatry;1999-02-01
3. Chlorpromazine and Diltiazem Effects in Muscle: Blockage of Acetylcholine-Evoked Contractures, Membrane Currents and Tracer Calcium Uptake;Pharmacology;1996
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