S100β promotes the extension of microtubule associated protein2 (MAP2)-immunoreactive neurites retracted after colchicine treatment in rat spinal cord culture-Reference-Cited by-同舟云学术

S100β promotes the extension of microtubule associated protein2 (MAP2)-immunoreactive neurites retracted after colchicine treatment in rat spinal cord culture

Published:1997-07 Issue:3 Volume:229 Page:212-214
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Nishi Mayumi,Kawata Mitsuhiro,Azmitia Efrain C

Publisher

Elsevier BV

Subject

General Neuroscience

Reference16 articles.

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2. S100β but not NGF, EGF, insulin or calmodulin is a CNS serotonergic growth factor;Azmitia;Brain Res.,1990

3. Azmitia, E.C. and Hou, X.P., Steroid regulation of neurotrophic activity: primary microcultures of mid brain raphe and hippocampus. In E.R. DeKloet and W. Sutanto (Eds.), Neurobiology of Steroids, Methods in Neuroscience, Vol. 22, Academic Press, San Diego, CA, 1994, pp. 359–371.

4. 5-HT1A agonist and dexamethasone reversal of parachloroamphetamine induced loss of MAP-2 and synaptophysin immunoreactivity in adult rat brain;Azmitia;Brain Res.,1995

5. Interactions between the microtubule-associated tau proteins and S100β regulate tau phosphorylation by the Ca2+/Calmodulin-dependent protein kinase II;Baudier;J. Biol. Chem.,1988

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