Acute effect of KA-672, a putative cognitive enhancer, on neurotransmitter receptor binding in mouse brain
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference15 articles.
1. The role of interactions between cholinergic system and other neuromodulatory systems in learning and memory;Decker;Synapse,1991
2. Noradrenergic modulation of cholinergic nucleus basalis neurons demonstrated by in vitro pharmacological and immunohistochemical evidence in the guinea-pig brain;Fort;Eur. J. Neurosci.,1995
3. Basal forebrain cholinergic immunolesion by 192IgG-saporin: evidence for a presynaptic location of subpopulations of α2-and β-adrenergic as well as 5-HT2A receptors on cortical cholinergic terminal;Heider;Neurochem. Res.,1997
4. KA-672 inhibits brain acetylcholinesterase in vitro but not in vivo;Hilgert;Neurosci. Lett.,1999
5. The putative cognitive enhancer KA-672.HCl is an uncompetitive voltage-dependent NMDA receptor antagonist;Lishko;NeuroReport,1998
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2. Cognitive Enhancers (Nootropics). Part 3: Drugs Interacting with Targets other than Receptors or Enzymes. Disease-modifying Drugs;Journal of Alzheimer's Disease;2013-02-06
3. Ensaculin (KA-672. HCl): A Multitransmitter Approach to Dementia Treatment;CNS Drug Reviews;2006-06-07
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