Elevated levels of Aβ1–40 and Aβ1–42 do not alter the binding sites of nicotinic receptor subtypes in the brain of APPswe and PS1 double transgenic mice-Reference-Cited by-同舟云学术

Elevated levels of Aβ1–40 and Aβ1–42 do not alter the binding sites of nicotinic receptor subtypes in the brain of APPswe and PS1 double transgenic mice

Published:2002-08 Issue:3 Volume:328 Page:269-272
ISSN:0304-3940
Container-title:Neuroscience Letters
language:en
Short-container-title:Neuroscience Letters

Author:

Marutle Amelia,Unger Christina,Hellström-Lindahl Ewa,Wang Jun,Puoliväli Jukka,Tanila Heikki,Nordberg Agneta,Zhang Xiao

Publisher

Elsevier BV

Subject

General Neuroscience

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3. Selective nicotinic receptor consequences in APPswe transgenic mice;Bednar;Mol. Cell. Neurosci.,2002

4. Accelerated amyloid deposition in the brains of transgenic mice coexpressing mutant presenilin 1 and amyloid precursor proteins;Borchelt;Neuron,1997

5. Beta-amyloid activates the mitogen-activated protein kinase cascade via hippocampal alpha7 nicotinic acetylcholine receptors: in vitro and in vivo mechanisms related to Alzheimer's disease;Dineley;J. Neurosci.,2001

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