Glutamate Release Induced by Activation of Glycine and GABA Transporters in Spinal Cord is Enhanced in a Mouse Model of Amyotrophic Lateral Sclerosis
Author:
Publisher
Elsevier BV
Subject
Toxicology,General Neuroscience
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1. Interactions Involving Glycine and Other Amino Acid Neurotransmitters: Focus on Transporter-Mediated Regulation of Release and Glycine–Glutamate Crosstalk;Biomedicines;2024-07-08
2. Characterization of the Mitochondrial Aerobic Metabolism in the Pre- and Perisynaptic Districts of the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis;Molecular Neurobiology;2018-04-14
3. Altered mechanisms underlying the abnormal glutamate release in amyotrophic lateral sclerosis at a pre-symptomatic stage of the disease;Neurobiology of Disease;2016-11
4. Exocytosis regulates trafficking of GABA and glycine heterotransporters in spinal cord glutamatergic synapses: a mechanism for the excessive heterotransporter-induced release of glutamate in experimental amyotrophic lateral sclerosis;Neurobiology of Disease;2015-02
5. Different presynaptic nicotinic receptor subtypes modulate in vivo and in vitro the release of glycine in the rat hippocampus;Neurochemistry International;2011-10
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