Pathophysiological mechanisms underlying increased anxiety after soman exposure: Reduced GABAergic inhibition in the basolateral amygdala-Reference-Cited by-同舟云学术

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Pathophysiological mechanisms underlying increased anxiety after soman exposure: Reduced GABAergic inhibition in the basolateral amygdala

Published:2014-09 Issue: Volume:44 Page:335-343
ISSN:0161-813X
Container-title:NeuroToxicology
language:en
Short-container-title:NeuroToxicology

Author:

Prager Eric M.,Pidoplichko Volodymyr I.,Aroniadou-Anderjaska Vassiliki,Apland James P.,Braga Maria F.M.

Funder

National Institutes of Health

Publisher

Elsevier BV

Subject

Toxicology,General Neuroscience

Reference68 articles.

1. Higher susceptibility of the ventral versus the dorsal hippocampus and the posteroventral versus anterodorsal amygdala to soman-induced neuropathology;Apland;Neurotoxicology,2010

2. Presynaptic facilitation of glutamate release in the basolateral amygdala: a mechanism for the anxiogenic and seizurogenic function of GluK1 receptors;Aroniadou-Anderjaska;Neuroscience,2012

3. Regional distribution of choline acetyltransferase and acetylcholinesterase within the amygdaloid complex and stria terminalis system;Ben-Ari;Brain Res,1977

4. The role of alpha7 and alpha4beta2 nicotinic receptors in the nicotine-induced anxiolytic effect in zebrafish;Bencan;Physiol Behav,2008

5. Core symptoms of posttraumatic stress disorder unimproved by alprazolam treatment;Braun;J Clin Psychiatry,1990

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