The GDF11-FTO-PPARγ axis controls the shift of osteoporotic MSC fate to adipocyte and inhibits bone formation during osteoporosis-Reference-Cited by-同舟云学术

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The GDF11-FTO-PPARγ axis controls the shift of osteoporotic MSC fate to adipocyte and inhibits bone formation during osteoporosis

Published:2018-12 Issue:12 Volume:1864 Page:3644-3654
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Shen Guang-Si,Zhou Hai-Bin,Zhang Hong,Chen Bin,Liu Zhi-Peng,Yuan Ye,Zhou Xiao-Zhong,Xu You-Jia

Funder

Jiangsu Province's Young Medical Talents Program

National Natural Science Foundation of China

Second Affiliated Hospital of Soochow University

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference46 articles.

1. Mechanisms of anabolic therapies for osteoporosis;Canalis;N. Engl. J. Med.,2007

2. Management of osteoporosis in elderly men;Gates;Maturitas,2011

3. Osteoporosis: now and the future;Rachner;Lancet,2011

4. Angiotensin II induces mitochondrial oxidative stress and mtDNA damage in osteoblasts by inhibiting SIRT1–FoxO3a–MnSOD pathway;Li;Biochem. Biophys. Res. Commun.,2014

5. Sirt3–MnSOD axis represses nicotine-induced mitochondrial oxidative stress and mtDNA damage in osteoblasts;Li;Acta Biochim. Biophys. Sin.,2015

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