Analysis of the functional consequences of lethal mutations in mitochondrial translational elongation factors-Reference-Cited by-同舟云学术

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Analysis of the functional consequences of lethal mutations in mitochondrial translational elongation factors

Published:2010-07 Issue:7-8 Volume:1802 Page:692-698
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Akama Kenta,Christian Brooke E.,Jones Christie N.,Ueda Takuya,Takeuchi Nono,Spremulli Linda L.

Funder

National Institutes of Health

JSPS

Naito Memorial Grant

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference35 articles.

1. Elongation factor Tu: a regulatory GTPase with an integrated effector;Sprinzl;Trends Biochem. Sci.,1994

2. Factors involved in the transfer of Aminoacyl-tRNA to the ribosome;Miller,1977

3. Distinct clinical phenotypes associated with a mutation in the mitochondrial translation elongation factor EF-Ts;Smeitink;Am. J. Hum. Genet.,2006

4. Crystal structure of the bovine mitochondrial elongation factor Tu:Ts complex;Jeppesen;J. Biol. Chem.,2005

5. Roles of residues in mammalian mitochondrial elongation factor Ts in the interaction with bacterial and mitochondrial elongation factor Tu;Zhang;J. Biol. Chem.,1998

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1. Molecular pathways in mitochondrial disorders due to a defective mitochondrial protein synthesis;Frontiers in Cell and Developmental Biology;2024-05-24

2. Reconstitution of mammalian mitochondrial translation system capable of correct initiation and long polypeptide synthesis from leaderless mRNA;Nucleic Acids Research;2020-12-09

3. Novel mutation in mitochondrial Elongation Factor EF-Tu associated to dysplastic leukoencephalopathy and defective mitochondrial DNA translation;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2017-04

4. The Protein Biosynthetic Machinery of Mitochondria;Encyclopedia of Cell Biology;2016

5. The presence of highly disruptive 16S rRNA mutations in clinical samples indicates a wider role for mutations of the mitochondrial ribosome in human disease;Mitochondrion;2015-11

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