Mutant huntingtin regulates EGF receptor fate in non-neuronal cells lacking wild-type protein-Reference-Cited by-同舟云学术

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Mutant huntingtin regulates EGF receptor fate in non-neuronal cells lacking wild-type protein

Published:2013-01 Issue:1 Volume:1832 Page:105-113
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Melone Mariarosa A.B.,Calarco Anna,Petillo Orsolina,Margarucci Sabrina,Colucci-D'Amato Luca,Galderisi Umberto,Koverech Guido,Peluso Gianfranco

Funder

Italian Ministry of University and Research

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference43 articles.

1. A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes. The Huntington's Disease Collaborative Research Group;AAVV;Cell,1993

2. Normal huntingtin function: an alternative approach to Huntington's disease;Cattaneo;Nat. Rev. Neurosci.,2005

3. Huntington's disease: new frontiers for molecular and cell therapy;Melone;Curr. Drug Targets,2005

4. New targets for therapy in polyglutamine (polyQ) expansion diseases;Perrone;Curr. Drug Ther.,2008

5. Generalized brain and skin proteasome inhibition in Huntington's disease;Seo;Ann. Neurol.,2004

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