Overexpression of Na+-HCO3– cotransporter contributes to the exacerbation of cardiac remodeling in mice with myocardial infarction by increasing intracellular calcium overload-Reference-Cited by-同舟云学术

Overexpression of Na+-HCO3– cotransporter contributes to the exacerbation of cardiac remodeling in mice with myocardial infarction by increasing intracellular calcium overload

Published:2020-03 Issue:3 Volume:1866 Page:165623
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Chen Zhenhuan,Chen Lu,Chen Kaitong,Lin Hairuo,Shen Mengjia,Chen Lin,Zhu Hailin,Zhu Yingqi,Wang Qiancheng,Xi Fang,Huang Xiaobo,Wang Yuegang,Liao Wangjun,Bin Jianping,Asakura Masanori,Liu Jie,Kitakaze Masafumi,Liao Yulin

Funder

National Natural Science Foundation of China

Guangzhou Municipal Science and Technology Project

Human Genome Tissue Engineering and Food Biotechnology

Natural Science Foundation of Guangdong Province

Key Research & Development Program of Guangzhou Regenerative Medical and Health Guangdong Laboratory

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference39 articles.

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4. Regulation of the cardiac sodium/bicarbonate cotransporter by angiotensin II: potential contribution to structural, ionic and electrophysiological myocardial remodelling;Aiello;Curr. Cardiol. Rev.,2013

5. Aldosterone stimulates the cardiac sodium/bicarbonate cotransporter via activation of the g protein-coupled receptor gpr30;De Giusti;J. Mol. Cell. Cardiol.,2015

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