HHV-6A infection dysregulates autophagy/UPR interplay increasing beta amyloid production and tau phosphorylation in astrocytoma cells as well as in primary neurons, possible molecular mechanisms linking viral infection to Alzheimer's disease-Reference-Cited by-同舟云学术

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HHV-6A infection dysregulates autophagy/UPR interplay increasing beta amyloid production and tau phosphorylation in astrocytoma cells as well as in primary neurons, possible molecular mechanisms linking viral infection to Alzheimer's disease

Published:2020-03 Issue:3 Volume:1866 Page:165647
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Romeo Maria Anele,Gilardini Montani Maria Saveria,Gaeta Aurelia,D'Orazi Gabriella,Faggioni Alberto,Cirone Mara

Funder

Istituto Pasteur-Fondazione Cenci Bolognetti

HHV-6 Foundation

Ministero dell’Istruzione, dell’Università e della Ricerca

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference38 articles.

1. Human herpesvirus-6 entry into the central nervous system through the olfactory pathway;Harberts;Proc. Natl. Acad. Sci. U. S. A.,2011

2. Variant-specific tropism of human herpesvirus 6 in human astrocytes;Donati;J. Virol.,2005

3. Glial cell responses to herpesvirus infections: role in defense and immunopathogenesis;Lokensgard;J. Infect. Dis.,2002

4. Infection with an endemic human herpesvirus disrupts critical glial precursor cell properties;Dietrich;J. Neurosci.,2004

5. Herpes simplex virus 1 tropism for human sensory ganglion neurons in the severe combined immunodeficiency mouse model of neuropathogenesis;Zerboni;J. Virol.,2013

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