Granulocytic myeloid-derived suppressor cells contribute to IFN-I signaling activation of B cells and disease progression through the lncRNA NEAT1-BAFF axis in systemic lupus erythematosus-Reference-Cited by-同舟云学术

Granulocytic myeloid-derived suppressor cells contribute to IFN-I signaling activation of B cells and disease progression through the lncRNA NEAT1-BAFF axis in systemic lupus erythematosus

Published:2020-01 Issue:1 Volume:1866 Page:165554
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Dong Guanjun,Yang Yonghong,Li Xuehui,Yao Xiaoying,Zhu Yuzhen,Zhang Hui,Wang Haiyan,Ma Qun,Zhang Junfeng,Shi Hui,Ning Zhaochen,Yan Fenglian,Zhai Weiwei,Dai Jun,Li Zhihua,Li Chunxia,Ming Jiankuo,Xue Qingjie,Meng Xiangzhi,Si Chuanping,Xiong Huabao

Funder

National Natural Science Foundation of China

Shandong Provincial Natural Science Foundation

Projects of Medical and Health Technology Development Program in Shandong Province

Jining Medical University

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference51 articles.

1. Distinct effector B cells induced by unregulated toll-like receptor 7 contribute to pathogenic responses in systemic lupus erythematosus;Jenks;Immunity,2018

2. Expanding the B cell-centric view of systemic lupus erythematosus;Morawski;Trends Immunol.,2017