Transient overexpression of Werner protein rescues starvation induced autophagy in Werner syndrome cells-Reference-Cited by-同舟云学术

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Transient overexpression of Werner protein rescues starvation induced autophagy in Werner syndrome cells

Published:2014-12 Issue:12 Volume:1842 Page:2387-2394
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Maity Jyotirindra,Bohr Vilhelm A.,Laskar Aparna,Karmakar Parimal

Funder

Council for Scientific and Industrial Research (CSIR)

Intramural program of the National Institute on Aging, NIH

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference45 articles.

1. RecQ helicases: caretakers of the genome;Hickson;Nat. Rev. Cancer,2003

2. RecQ helicases in DNA repair, recombination, and replication;Croteau;Annu. Rev. Biochem.,2014

3. Current advances in unraveling the function of the Werner syndrome protein;Ozgenc;Mutat. Res.,2005

4. Werner syndrome protein characterization of the integral 3′-5′ DNA exonuclease;Kamath-Loeb;J. Biol. Chem.,1998

5. The RecQ helicase WRN is required for normal replication fork progression after DNA damage or replication fork arrest;Sidorova;Cell Cycle,2008

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4. Crosstalk among DNA Damage, Mitochondrial Dysfunction, Impaired Mitophagy, Stem Cell Attrition, and Senescence in the Accelerated Ageing Disorder Werner Syndrome;Cytogenetic and Genome Research;2021

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