The damage-associated molecular pattern HMGB1 is released early after clinical hepatic ischemia/reperfusion

Author:

van Golen Rowan F.,Reiniers Megan J.,Marsman Gerben,Alles Lindy K.,van Rooyen Derrick M.,Petri Björn,Van der Mark Vincent A.,van Beek Adriaan A.,Meijer Ben,Maas Martinus A.,Zeerleder Sacha,Verheij Joanne,Farrell Geoffrey C.,Luken Brenda M.,Teoh Narci C.,van Gulik Thomas M.,Murphy Michael P.,Heger Michal

Funder

Academic Medical Center

Stichting Nationaal Fonds Tegen Kanker

Phospholipid Research Center

Nijbakker-Morra Foundation

Stichting Technologische Wetenschap

Dutch Cancer Society

CFI

University of Calgary

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference54 articles.

1. V. Notes on the arrest of hepatic hemorrhage due to trauma;Pringle;Ann. Surg.,1908

2. The sterile immune response during hepatic ischemia/reperfusion;van Golen;Cytokine Growth Factor Rev.,2012

3. How much ischemia can the liver tolerate during resection?;van Riel;Hepatobiliary Surg. Nutr.,2016

4. Mechanistic overview of reactive species-induced degradation of the endothelial glycocalyx during hepatic ischemia/reperfusion injury;van Golen;Free Radic. Biol. Med.,2012

5. A unifying mechanism for mitochondrial superoxide production during ischemia-reperfusion injury;Chouchani;Cell Metab.,2016

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