Endogenous osteopontin induces myocardial CCL5 and MMP-2 activation that contributes to inflammation and cardiac remodeling in a mouse model of chronic Chagas heart disease-Reference-Cited by-同舟云学术

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Endogenous osteopontin induces myocardial CCL5 and MMP-2 activation that contributes to inflammation and cardiac remodeling in a mouse model of chronic Chagas heart disease

Published:2018-01 Issue:1 Volume:1864 Page:11-23
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Caballero Eugenia Pérez,Santamaría Miguel H.,Corral Ricardo S.

Funder

Iberoamerican Science Consortium

National Research Council

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference66 articles.

1. Chagas disease cardiomyopathy: current concepts of an old disease;Bilate;Rev. Inst. Med. Trop. Sao Paulo,2008

2. Chagas disease: present status of pathogenic mechanisms and chemotherapy;Maya;Biol. Res.,2010

3. Association of an increase in CD8+ T cells with the presence of Trypanosoma cruzi antigens in chronic, human, chagasic myocarditis;Higuchi;Am. J. Trop. Med. Hyg.,1997

4. Parasite persistence in the aetiology of Chagas disease;Tarleton;Int. J. Parasitol.,2001

5. Regulated on activation, normal T cell expressed and secreted (RANTES) antagonist (Met-RANTES) controls the early phase of Trypanosoma cruzi-elicited myocarditis;Marino;Circulation,2004

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