Helicobacter pylori chronic infection and mucosal inflammation switches the human gastric glycosylation pathways

Author:

Magalhães Ana,Marcos-Pinto Ricardo,Nairn Alison V.,dela Rosa Mitche,Ferreira Rui M.ORCID,Junqueira-Neto Susana,Freitas Daniela,Gomes Joana,Oliveira Patrícia,Santos Marta R.,Marcos Nuno T.,Xiaogang Wen,Figueiredo Céu,Oliveira Carla,Dinis-Ribeiro Mário,Carneiro FátimaORCID,Moremen Kelley W.,David Leonor,Reis Celso A.

Funder

FCT, the Portuguese Foundation for Science and Technology

FEDER funds through the Operational Programme for Competitiveness Factors-COMPETE

National Funds through the FCT-Foundation for Science and Technology

Seventh Framework Programme GastricGlycoExplorer project

FCT

POPH (Programa Operacional Potencial Humano)

FSE (Fundo Social Europeu)

EMBO

NIH

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference62 articles.

1. Carcinogenesis of Helicobacter pylori;Correa;Gastroenterology,2007

2. GLOBOCAN 2012 v1.0, Cancer Incidence and Mortality Worldwide;Ferlay,2013

3. Helicobacter pylori and gastric cancer: factors that modulate disease risk;Wroblewski;Clin. Microbiol. Rev.,2010

4. Helicobacter pylori CagA and gastric Cancer: a paradigm for hit-and-run carcinogenesis;Hatakeyama;Cell Host & Microbe,2014

5. Infection with Helicobacter pylori strains possessing cagA is associated with an increased risk of developing adenocarcinoma of the stomach;Blaser;Cancer Res.,1995

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