Farnesoid X receptor overexpression prevents hepatic steatosis through inhibiting AIM2 inflammasome activation in nonalcoholic fatty liver disease-Reference-Cited by-同舟云学术

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Farnesoid X receptor overexpression prevents hepatic steatosis through inhibiting AIM2 inflammasome activation in nonalcoholic fatty liver disease

Published:2024-02 Issue:2 Volume:1870 Page:166930
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Xu Shuai,Kong Lina,Li Lin,Wang Changyuan,Gu Jiangning,Luo Haifeng,Meng Qiang

Funder

Dalian Medical University

Natural Science Foundation of Liaoning Province

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference57 articles.

1. Genetic and epigenetic factors determining NAFLD risk;Jonas;Mol. Metab.,2021

2. Molecular pathways of nonalcoholic fatty liver disease development and progression;Bessone;Cell. Mol. Life Sci.,2019

3. Hepatocyte-specific Keap1 deletion reduces liver steatosis but not inflammation during non-alcoholic steatohepatitis development;Ramadori;Free Radic. Biol. Med.,2016

4. Role of gut microbiota and oxidative stress in the progression of non-alcoholic fatty liver disease to hepatocarcinoma: current and innovative therapeutic approaches;Borrelli;Redox Biol.,2018

5. The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome;Lim;Nat. Rev. Gastroenterol. Hepatol.,2010

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