NPC1-like phenotype, with intracellular cholesterol accumulation and altered mTORC1 signaling in models of Parkinson's disease-Reference-Cited by-同舟云学术

NPC1-like phenotype, with intracellular cholesterol accumulation and altered mTORC1 signaling in models of Parkinson's disease

Published:2024-02 Issue:2 Volume:1870 Page:166980
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Caria Inês,Nunes Maria João,Ciraci Viviana,Carvalho Andreia Neves,Ranito Catarina,Santos Susana G.,Gama Maria João,Castro-Caldas Margarida,Rodrigues Cecília M.P.,Ruas Jorge L.,Rodrigues Elsa

Funder

Fundação para a Ciência e a Tecnologia

Swedish Research Council

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference69 articles.

1. Brain cell type-specific cholesterol metabolism and implications for learning and memory;Li;Trends Neurosci.,2022

2. Parkinson disease;Poewe;Nat. Rev. Dis. Primers.,2017

3. Parkinson’s disease brain mitochondrial complex I has oxidatively damaged subunits and is functionally impaired and misassembled;Keeney;J. Neurosci.,2006

4. Mitochondrial dysfunction and oxidative stress in Parkinson’s disease;Subramaniam;Prog. Neurobiol.,2013

5. MPTP mouse models of Parkinson’s disease: an update;Meredith;J. Parkinsons Dis.,2011

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Upregulation of cholesterol synthesis by lysosomal defects requires a functional mitochondrial respiratory chain;Journal of Biological Chemistry;2024-07

2. Up-regulation of cholesterol synthesis by lysosomal defects requires a functional mitochondrial respiratory chain;2024-03-07