Enhanced parkin levels favor ER-mitochondria crosstalk and guarantee Ca2+ transfer to sustain cell bioenergetics-Reference-Cited by-同舟云学术

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Enhanced parkin levels favor ER-mitochondria crosstalk and guarantee Ca2+ transfer to sustain cell bioenergetics

Published:2013-04 Issue:4 Volume:1832 Page:495-508
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Calì Tito,Ottolini Denis,Negro Alessandro,Brini Marisa

Funder

Italian Ministry of University and Research

University of Padova

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference68 articles.

1. Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases;Lin;Nature,2006

2. Mitochondria in the aetiology and pathogenesis of Parkinson's disease;Schapira;Lancet Neurol.,2008

3. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy;Narendra;J. Cell Biol.,2008

4. PINK1-dependent recruitment of Parkin to mitochondria in mitophagy;Vives-Bauza;Proc. Natl. Acad. Sci. U. S. A.,2010

5. PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy;Matsuda;J. Cell Biol.,2010

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