The ALK-1/Smad1 pathway in cardiovascular physiopathology. A new target for therapy?-Reference-Cited by-同舟云学术

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The ALK-1/Smad1 pathway in cardiovascular physiopathology. A new target for therapy?

Published:2013-10 Issue:10 Volume:1832 Page:1492-1510
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

González-Núñez María,Muñoz-Félix José M.,López-Novoa José M.

Funder

Ministerio de Ciencia e Innovación of Spain

Junta de Castilla y Leon

Red de Investigación Cooperativa en Enfermedades Renales (REDINREN

Instituto de Salud Carlos III

Fondo Social Europeo

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference249 articles.

1. How cells read TGF-beta signals;Massague;Nat. Rev. Mol. Cell Biol.,2000

2. Balancing the activation state of the endothelium via two distinct TGF-beta type I receptors;Goumans;EMBO J.,2002

3. TGF-beta receptor function in the endothelium;Lebrin;Cardiovasc. Res.,2005

4. L- and S-endoglin differentially modulate TGFbeta1 signaling mediated by ALK1 and ALK5 in L6E9 myoblasts;Velasco;J. Cell Sci.,2008

5. Genetic and pharmacological targeting of activin receptor-like kinase 1 impairs tumor growth and angiogenesis;Cunha;J. Exp. Med.,2010

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