S100A4 mediates the accumulation and functions of myeloid-derived suppressor cells via GP130/JAK2/STAT3 signaling in acute myeloid leukemia-Reference-Cited by-同舟云学术

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S100A4 mediates the accumulation and functions of myeloid-derived suppressor cells via GP130/JAK2/STAT3 signaling in acute myeloid leukemia

Published:2025-01 Issue:1 Volume:1871 Page:167498
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Peng Yuhui,Zhang Jian,Zhang Ting,Wang Chanjuan,Bai Jingdi,Li Yi,Duan Juanjuan,Fan Daogui,Fu Wenli,Liang Xinming,Xie Xin,Qi Xiaolan,Hong Wei,He Yan,Wu ChangXue,Zhou Jing,Chen Pingping,Zeng Hongmei,Dai Yun,Yu Wenfeng,Bai Hua,Guo Pengxiang,Zeng Zhu,Zhang Qifang

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Reference53 articles.

1. Acute myeloid leukemia in the elderly: therapeutic options and choice;Webster;Leuk. Lymphoma,2018

2. Treatment of relapsed/refractory acute myeloid leukemia;Bose;Curr. Treat. Options in Oncol.,2017

3. Treatment of relapsed/refractory acute myeloid leukaemia in adults;Rashidi;Br. J. Haematol.,2018

4. The role of the immunosuppressive microenvironment in acute myeloid leukemia development and treatment;Isidori;Expert Rev. Hematol.,2014

5. Immunosuppression and Immunotargeted therapy in acute myeloid leukemia - the potential use of checkpoint inhibitors in combination with other treatments;Leufven;Curr. Med. Chem.,2019

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