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Mitochondrial dysfunction in amyotrophic lateral sclerosis

Published:2010-01 Issue:1 Volume:1802 Page:45-51
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Shi Ping,Gal Jozsef,Kwinter David M.,Liu Xiaoyan,Zhu Haining

Funder

NIH

H.Z

NIH/NCRR Center of Biomedical Research Excellence in the Molecular Basis of Human Disease

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference116 articles.

1. Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis;Rosen;Nature,1993

2. Unraveling the mechanisms involved in motor neuron degeneration in ALS;Bruijn;Annu. Rev. Neurosci.,2004

3. TARDBP mutations in individuals with sporadic and familial amyotrophic lateral sclerosis;Kabashi;Nat. Genet,2008

4. TDP-43 mutations in familial and sporadic amyotrophic lateral sclerosis;Sreedharan;Science,2008

5. Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6;Vance;Science,2009

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