Loss of synaptic zinc transport in progranulin deficient mice may contribute to progranulin-associated psychopathology and chronic pain-Reference-Cited by-同舟云学术

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Loss of synaptic zinc transport in progranulin deficient mice may contribute to progranulin-associated psychopathology and chronic pain

Published:2017-11 Issue:11 Volume:1863 Page:2727-2745
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Hardt Stefanie,Heidler Juliana,Albuquerque Boris,Valek Lucie,Altmann Christine,Wilken-Schmitz Annett,Schäfer Michael K.E.,Wittig Ilka,Tegeder Irmgard

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference79 articles.

1. Advances in understanding the molecular basis of frontotemporal dementia;Rademakers;Nat. Rev. Neurol.,2012

2. Possible involvement of lysosomal dysfunction in pathological changes of the brain in aged progranulin-deficient mice;Tanaka;Acta Neuropathol. Commun.,2014

3. Accelerated lipofuscinosis and ubiquitination in granulin knockout mice suggest a role for progranulin in successful aging;Ahmed;Am. J. Pathol.,2010

4. Progranulin overexpression in sensory neurons attenuates neuropathic pain in mice: role of autophagy;Altmann;Neurobiol. Dis.,2016

5. Rescue of progranulin deficiency associated with frontotemporal lobar degeneration by alkalizing reagents and inhibition of vacuolar ATPase;Capell;J. Neurosci.,2011

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