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S100B promotes injury-induced vascular remodeling through modulating smooth muscle phenotype

  • Published:2017-11 Issue:11 Volume:1863 Page:2772-2782
  • ISSN:0925-4439
  • Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
  • language:en
  • Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Cao Teng,Zhang Lei,Yao Ling-Ling,Zheng Fei,Wang Lu,Yang Jian-Ye,Guo Ling-Yun,Li Xing-Yuan,Yan Yu-Wen,Pan Ya-Mu,Jiang Miao,Chen Long,Tang Jun-Ming,Chen Shi-You,Wang Jia-Ning

Funder

National natural Science Foundation of China

National natural Science Foundation of Hubei

Hubei University of Medicine Innovative team

National Institutes of Health

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference25 articles.

1. Coronary artery disease: interventional narrowing of the coronary sinus in refractory angina;Lim;Nat. Rev. Cardiol.,2015

2. Vascular smooth muscle cell in atherosclerosis;Chistiakov;Acta Physiol (Oxf).,2015

3. S100B's double life: Intracellular regulator and extracellular signal;Donato;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research,1793

4. S100: a multigenic family of calcium-modulated proteins of the EF-hand type with intracellular and extracellular functional roles;Donato;Int. J. Biochem. Cell Biol.,2001

5. Regulation of the S100B gene by α1-adrenergic stimulation in cardiac myocytes;Tsoporis;Am. J. Physiol. Heart Circ. Physiol.,2003
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